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This review provides a critical and translational analysis of GSH-responsive nanomedicine, highlighting chemical strategies such as disulfide/diselenide linkages, transition metal systems, and GSH-activated prodrugs

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Journal of Dermatologic Science and Cosmetic Technology

2000;2000(4):CD000950

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Because cancer cells hijack the protective capability of NRF2 to promote drug resistance and proliferation, specific NRF2 inhibitors would be desirable for the treatment of cancers that have constitutively active NRF2